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The Missing Vitamins In Mystery Diseases

The Missing Vitamins In Mystery Diseases

Autoimmune diseases are on the rise, many without known medical causes or cures. There’s also a category of diseases known as diagnoses of exclusion, where testing for common diseases with your symptoms are normal, so the diagnosis is made based on a process of elimination. These diseases include Parkinson’s, Migraine, Fibromyalgia, Chronic Fatigue Immune Deficiency Syndrome (CFIDS), and more. There are key vitamin and nutrient deficiencies found in many of these conditions, and correcting them can produce dramatic results, providing one more piece of the puzzle in putting your health back together. Continue reading to discover the missing vitamins found in mystery diseases.

Migraine Relief

Migraine is more than just a headache. Symptoms are debilitating and range from the classic nausea, light sensitivity and extreme headache to even mimic a stroke with trouble finding words and slurred speech, numbness and difficulty moving arms and legs.

Riboflavin (Vitamin B2) has been shown in studies to decrease frequency and number of migraines. It’s found in small amounts in a variety of foods including meat, dairy, legumes, nuts, eggs and leafy greens, but is easily destroyed by exposure to light or an alkaline environment. The effective supplement dose is 400mg daily, which may take 3 months to see results.

Low magnesium in the brain has been measured during a migraine attack, and many migraine sufferers are found to be magnesium deficient overall, especially those with menstrual migraine. Magnesium relaxes smooth muscles, and high dose supplementation of 600mg or more has been shown to prevent migraine attacks. Magnesium oxide and citrate forms are known to cause diarrhea at high doses. The malate, threonate and glycinate forms are better tolerated at the dose needed to prevent migraine.

Coenzyme Q10 (CoQ10) is a strong antioxidant involved in the body’s energy production through the ATP molecule. An ongoing dose of 150mg daily can decrease the number of days and intensity of migraine attacks. The effect is cumulative and may take 3 months before results are seen.

Help with Hashimoto’s Fatigue

Hashimoto’s Autoimmune Thyroiditis is the most common cause of hypothyroidism. The fatigue can be crippling despite thyroid meds and a grain free diet. Low thiamine (Vitamin B1) can be the cause of your fatigue. The most common source of this vitamin is fortified grains, so the grain free diet that does so much good for you can also lead to thiamine deficiency. Supplementing with 600mg daily of benfotiamine can relieve fatigue in less than a week.

Slowing the Progression of Parkinson’s

Parkinson’s is a disease that progresses to complete disability, and the drug side effects can be worse than the disease itself. Glutathione is the second strongest antioxidant in the body and is found to be deficient in the brains of Parkinson’s patients. Glutathione IV therapy was made famous through David Perlmutter, MD, whose remarkable results restoring mobility to severe Parkinson’s sufferers brought new hope and research for a disease with very few answers.

Glutathione IV dosing ranges from 600mg twice daily for 30 days, to an ongoing biweekly dose starting at 900mg and working up to 3000mg. Liposomal glutathione, S-Acetyl Glutathione or sublingual glutathione is often supplemented at home between IV doses to keep levels up.

Testing for Vitamin Deficiencies

Your regular health care provider can do blood testing for some vitamin deficiencies, but these usually only show severe deficiency. Your body may have a functional deficiency, where you have enough to stay alive but not enough to overcome disease or achieve optimal health. There is more comprehensive testing available through labs like Spectracell or Genova Diagnostics, if you feel you need testing. You may still see positive results from supplementing even when all of these tests look normal.

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Prog Neuropsychopharmacol Biol Psychiatry. 1996 Oct;20(7):1159-70. Reduced intravenous glutathione in the treatment of early Parkinson’s disease.
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